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Webster New World Dictionary Second College Edition Thumb Index(Hardcover, 1986)

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Item specifics

Condition:
Good: A book that has been read but is in good condition. Very minimal damage to the cover including ...
Series:
Webster's New World
Publication Year:
1986
Type:
Monolingual Dictionary
Format:
Hardcover
Language:
English
Publication Name:
College dictionary by webster's new world
Author:
Webster
Features:
Thumb Indexed, Illustrated
Publisher:
Prentice Hall Press
Country/Region of Manufacture:
United States
Modified Item:
No
Subject:
Dictionary
ISBN:
Does not apply







Webster New World Dictionary Second College Edition Thumb Index(Hardcover, 1986)

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a leading international journal from BMJ and BSG, publishes cutting-edge gastroenterology and hepatology research and reviews

Impact Factor: 23.059
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Gut is a Plan S compliant Transformative Journal.

Gut is a leading international journal in gastroenterology and hepatology and has an established reputation for publishing first class clinical research of the alimentary tract, the liver, biliary tree and pancreas. Gut delivers up-to-date, authoritative, clinically oriented coverage in all areas of gastroenterology and hepatology. Regular features include articles by leading authorities describing novel mechanisms of disease and new management strategies, both diagnostic and therapeutic, likely to impact on clinical practice within the foreseeable future.

Gut is an official journal of the British Society of Gastroenterology and has two companion titles: Frontline Gastroenterology for education and practice and BMJ Open Gastroenterology for sound science clinical research.

Editor-in-Chief: Professor Emad El-Omar, University of New South Wales, Sydney, Australia
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Synergistic targeting and resistance to PARP inhibition in pancreatic cancer

To cite: Gout J, Perkhofer L, Morawe M, et al. Gut 2021;70:743-760.

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Objective: ATM serine/threonine kinase (ATM) is the most frequently mutated DNA damage response gene, involved in homologous recombination (HR), in pancreatic ductal adenocarcinoma (PDAC).

Conclusion: Analysis of the molecular susceptibilities triggered by ATM deficiency in PDAC allow elaboration of an efficient mutation-specific combinational therapeutic approach that can be also implemented in a genotype-independent manner by ATM inhibition.

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